Myocardial Injury and Left Ventricular Performance After Subarachnoid Hemorrhage
نویسندگان
چکیده
منابع مشابه
Myocardial injury and left ventricular performance after subarachnoid hemorrhage.
BACKGROUND AND PURPOSE Electrocardiographic abnormalities and elevations of the creatine kinase myocardial isoenzyme (CK-MB) occur frequently after subarachnoid hemorrhage. In some patients, a reversible and presumably neurogenic form of left ventricular dysfunction is demonstrated by echocardiography. It is not known whether cardiac injury of this type adversely affects cardiovascular hemodyna...
متن کاملRegional myocardial perfusion after experimental subarachnoid hemorrhage.
BACKGROUND AND PURPOSE The pathophysiology of cardiac injury after subarachnoid hemorrhage (SAH) remains controversial. Data from animal models suggest that catecholamine-mediated injury is the most likely cause of cardiac injury after SAH. However, researchers also have proposed myocardial ischemia to be the underlying cause, as a result of coronary artery disease, coronary artery spasm, or hy...
متن کاملAcute neurocardiogenic injury after subarachnoid hemorrhage.
BACKGROUND Left ventricular (LV) systolic dysfunction has been reported in humans with subarachnoid hemorrhage (SAH), and its underlying pathophysiology remains controversial. Possible mechanisms include myocardial ischemia versus excessive catecholamine release from sympathetic nerve terminals. METHODS AND RESULTS For 38 months, echocardiography and myocardial scintigraphy with technetium se...
متن کاملPredictors of neurocardiogenic injury after subarachnoid hemorrhage.
BACKGROUND AND PURPOSE Subarachnoid hemorrhage (SAH) frequently results in myocardial necrosis with release of cardiac enzymes. Historically, this necrosis has been attributed to coronary artery disease, coronary vasospasm, or oxygen supply-demand mismatch. Experimental evidence, however, indicates that excessive release of norepinephrine from the myocardial sympathetic nerves is the most likel...
متن کاملTransient Left Ventricular Apical Ballooning Syndrome and Cardiac Dysfunction after Subarachnoid Hemorrhage: Similar Clinical Entities?
Universally accepted diagnostic criteria for transient left ventricular apical ballooning syndrome are not yet available and the cardiac dysfunction complicating subarachnoid hemorrhage drops several hints of discussion about the clinical and pathophysiological similarities with the “typical” transient left ventricular apical ballooning syndrome. We suggest that, in the light of clinical and pa...
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ژورنال
عنوان ژورنال: Stroke
سال: 1999
ISSN: 0039-2499,1524-4628
DOI: 10.1161/01.str.30.4.780